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Much of our understanding of the pathobiology of TBI have arisen from animal models that mimic features of human TBI

In medicine, when a prescribed substance is believed to have caused an adverse effect (such as weight gain), its reported - meaning its publicly registered so physicians and scientists can learn about it and be on the lookout for similar problems

One of the most consistent findings is a reduction in circulating free T3 (fT3) levels, often without a corresponding rise in TSH, suggesting a non-pathological downregulation of thyroid activity [10,12]